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2005). These final results were not stunning if we take into consideration that PEA is a poorly drinking water‐soluble compound, which could Restrict its oral absorption and bioavailability, Nonetheless they could also advise a short‐lived action of PEA, in arrangement with The truth that this compound is degraded by two distinct hydrolases, th
2015). These scientific tests, taken alongside one another, suggest that, Despite the fact that a direct activation of GPR55 or PPAR‐α happens, PEA can create its anti‐inflammatory action within the intestine also via indirect activation of CB1 and CB2 receptors, likely as a result of the ability of this compound to potentiate the action of en
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